Porphyria Cutanea Tarda Treated by Repeated Venesection: Clinical and Biochemical Response
                    
                        
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                    چکیده
منابع مشابه
Clinical spectrum of porphyria cutanea tarda.
Correspondence: Giovanni Borroni, Clinica Dermatologica, IRCCS Policlinico S. Matteo, 27100 Pavia, Italy. Phone: international +39-0382-503494 – Fax. international +390382-526379. The term porphyria cutanea tarda (PCT) refers to a group of disorders biochemically charcaterized by reduced activity of uroporphyrinogen decarboxylase in liver and, in familial cases, in a number of tissues and cells...
متن کاملFamilial porphyria cutanea tarda.
Semiautomated Detection of Human Papifiomavirus DNA of High and Low Oncogenic Potential in Cervical Smears. George Terry, Linda Ho, Anne Szarewski, Jack Cuzick 1890 Immunoassay with Recombinant Trypanosoma cruzi Antigens Potentially Useful for Screening Donated Blood and Diagnosing Chagas Disease. Ana C. Pastini, Sergio R. Iglesias, Valentina C. Carri carte, Marcelo E. Guerin, Daniel 0. Sanchez...
متن کاملPorphyria cutanea tarda and melioidosis.
Porphyria cutanea tarda is a metabolic disorder in the haem biosynthetic pathway. It includes a heterogeneous group of conditions, which may be inherited or, more commonly, acquired. Although porphyria cutanea tarda presents with cutaneous lesions only, it is often associated with systemic disease. A 64-year-old Chinese patient, who developed sporadic porphyria cutanea tarda 1 year after the di...
متن کاملPorphyria Cutanea Tarda is a Biochemical and Not Histological Diagnosis
Porphyria cutanea tarda (PCT), the most common human porphyria, is acquired due to deficient activity of hepatic uroporphyrinogen decarboxylase (UROD) in the heme biosynthetic pathway, secondary to production of its inhibitor uroporphomethene, which is produced in the presence of mild to moderate iron overload [1-4]. Decreased activity of UROD leads to increased accumulation of uroporphyrinogen...
متن کامل[Porphyria cutanea tarda and hemochromatosis in Spain].
To the Editor: We present the case of a 31-year-old woman with no relevant medical history, who consulted because of lesions on the backs of the hands related to minor traumas; the lesions worsened in the summer leaving residual hyperpigmentation (Figure). The patient had phototype III skin and displayed discrete hypertrichosis in the zygomatic areas. Photosensitive dermatosis was suspected and...
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ژورنال
عنوان ژورنال: Proceedings of the Royal Society of Medicine
سال: 1969
ISSN: 0035-9157
DOI: 10.1177/003591576906200629